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Physalaemin

● Preclinical
Physalaemin
Also known as: PHY, South American Frog Tachykinin
Page last reviewed

Quick Summary

Physalaemin is an 11-residue tachykinin peptide first isolated from the skin of the South American frog Physalaemus fuscumaculatus. It shares the conserved tachykinin C-terminal sequence Phe-X-Gly-Leu-Met-NH2 with substance P, eledoisin, and kassinin.

Neuropeptide / Tachykinin Preclinical
Physalaemin is an 11-residue tachykinin peptide first isolated from the skin of the South American frog Physalaemus fuscumaculatus. It shares the conserved tachykinin C-terminal sequence Phe-X-Gly-Leu-Met-NH2 with substance P, eledoisin, and kassinin. Physalaemin was the first tachykinin of amphibian origin to be characterized and has been widely used as a reference ligand in tachykinin receptor pharmacology.
Storage Stability
Lyophilized
1–2 years (-20°C)
Reconstituted
~30 days (2–8°C)
Room temp
Avoid

Mechanism of Action

NK1 Receptor Preference

Physalaemin preferentially activates NK1 receptors (the substance P-preferring receptor), leading to Gq activation, PLC stimulation, and intracellular calcium mobilization. In the gastrointestinal tract, NK1 activation induces intestinal smooth muscle contraction and increases secretion. In the vasculature, it causes endothelium-dependent vasodilation.

Salivary and Exocrine Secretion

Like eledoisin and substance P, physalaemin is a potent stimulator of salivary gland secretion. It also stimulates pancreatic exocrine secretion through NK1 receptors on acinar cells. These secretagogue properties have been exploited in experimental models of exocrine gland function.


Research Summary

Tachykinin Receptor Pharmacology

Preclinical

Physalaemin has been used as a reference agonist in comparative tachykinin receptor studies. Its potency profile across NK1, NK2, and NK3 receptors helped establish the selectivity requirements for each receptor subtype. Structure-activity relationship studies using physalaemin analogs identified the key C-terminal residues responsible for receptor recognition.

Antimicrobial Properties

Preclinical

Like many amphibian skin peptides, physalaemin displays secondary antimicrobial activity against bacteria and fungi at higher concentrations. This activity is related to its amphipathic helical character in membrane environments, separate from its receptor-mediated pharmacological actions.


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Research Protocols

GoalDoseFrequencyRoute
NK receptor binding assays1-1000 nMSingleReceptor binding / cell assay
Smooth muscle contraction10-100 nMSingleOrgan bath preparation

Research use only. No therapeutic applications.


Interactions

Synergy
Substance P
Both NK1 preferring tachykinins; additive smooth muscle effects
Synergy
Related amphibian tachykinin; complementary receptor profiles

Safety Profile

Physalaemin produces hypotension through vasodilation and may cause bronchoconstriction via airway smooth muscle NK1 activation. Not used therapeutically in humans. Used solely as a pharmacological research tool.


References

  • [1]Anastasi A et al. (1964). Isolation and structure of physalaemin, the main active polypeptide of the skin of Physalaemus fuscumaculatus. Archives of Biochemistry and Biophysics, 108, 341-348.
  • [2]Regoli D and Barabe J (1980). Pharmacology of bradykinin and related kinins. Pharmacological Reviews, 32(1), 1-46.
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Verified Scientific Data Last audited:
Data Sources & External References
Source: peer-reviewed literature  ·  Domain: ascendpeptide.org

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