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Kassinin

● Preclinical
Kassinin
Also known as: KAS, NK2 Preferring Tachykinin, African Frog Tachykinin
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Quick Summary

Kassinin is a tachykinin peptide isolated from the skin of the African frog Kassina senegalensis. It preferentially activates NK2 receptors, distinguishing it from physalaemin (NK1) and making it valuable in delineating NK2 receptor pharmacology.

Neuropeptide / Tachykinin Preclinical
Kassinin is a tachykinin peptide isolated from the skin of the African frog Kassina senegalensis. It preferentially activates NK2 receptors, distinguishing it from physalaemin (NK1) and making it valuable in delineating NK2 receptor pharmacology. Kassinin was important in establishing that multiple tachykinin receptor subtypes exist with distinct pharmacological profiles, which eventually led to the development of NK2 antagonists for respiratory and GI conditions.
Storage Stability
Lyophilized
1–2 years (-20°C)
Reconstituted
~30 days (2–8°C)
Room temp
Avoid

Mechanism of Action

NK2 Receptor Selectivity

Kassinin has approximately 10-fold selectivity for NK2 receptors over NK1 receptors. NK2 is expressed in smooth muscle of the bronchi, gastrointestinal tract, and urinary tract. Gq-coupled NK2 activation causes bronchoconstriction and intestinal motility changes. This receptor preference made kassinin a key tool in establishing NK2 as a distinct receptor subtype.

Bronchomotor Effects

Kassinin constricts bronchial smooth muscle via NK2 activation, mimicking aspects of airway hyperresponsiveness seen in asthma. Studies using kassinin and NK2 antagonists helped establish the role of tachykinin signaling in bronchospasm, contributing to the development of NK2 antagonists for asthma therapy.


Research Summary

NK2 Receptor Pharmacology

Preclinical

Kassinin served as a pivotal tool in the discovery of NK2 receptor selectivity. Comparative studies using kassinin, substance P, and other tachykinins across multiple tissue preparations revealed differential potency profiles that could not be explained by a single receptor class, leading to the NK1/NK2/NK3 classification.

GI Motility Research

Preclinical

In gastrointestinal pharmacology, kassinin activates NK2 on intestinal smooth muscle to stimulate propulsive contractions. This has been used to model constipation-predominant bowel disorders and to evaluate NK2 antagonists as potential motility agents.


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Research Protocols

GoalDoseFrequencyRoute
NK2 receptor binding1-1000 nMSingleBinding assay
Bronchoconstriction model10-300 nMSingleIsolated trachea preparation

Research tool only. Not used therapeutically.


Interactions

Antagonism
NK2 antagonists (nepadutant)
Competitive inhibition; kassinin used as reference agonist
Complementary
NK1 vs NK2 preference allows receptor subtype discrimination

Safety Profile

Kassinin causes bronchoconstriction and blood pressure changes through NK2 and NK1 activation. Not used therapeutically. Research applications are limited to in vitro pharmacology and animal models.


References

  • [1]Anastasi A et al. (1977). Kassinin, a tachykinin dodecapeptide occurring in the skin of the African frog Kassina senegalensis. Identification and pharmacological properties. Experientia, 33(7), 857-858.
  • [2]Maggi CA (1995). The mammalian tachykinin receptors. General Pharmacology, 26(5), 911-944.
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Data Sources & External References
Source: peer-reviewed literature  ·  Domain: ascendpeptide.org

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