📚 Wiki Hormonal & Reproductive Kisspeptin-54

Kisspeptin-54

◎ Phase 2
Kisspeptin-54 (Metastin)
Also known as: KP-54, Metastin, Kisspeptin Full-Length
Brand names: Kisspeptin-54 (research grade), Metastin
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Quick Summary

Kisspeptin-54 (KP-54, also called metastin) is the 54-amino acid full-length processed form of the KISS1 gene product. It is the predominant form in the hypothalamus and pituitary.

Neuropeptide / Reproductive Hormone Phase 2
Kisspeptin-54 (KP-54, also called metastin) is the 54-amino acid full-length processed form of the KISS1 gene product. It is the predominant form in the hypothalamus and pituitary. Kisspeptin-54 activates the KISS1R (GPR54) receptor on GnRH neurons, triggering GnRH pulse generation and downstream LH/FSH release. It is being evaluated in clinical trials for infertility treatment and hypothalamic amenorrhea.
Storage Stability
Lyophilized
1–2 years (-20°C)
Reconstituted
~30 days (2–8°C)
Room temp
Avoid

Mechanism of Action

KISS1R/GPR54 Activation

Kisspeptin-54 binds KISS1R (GPR54), a Gq/G11-coupled GPCR expressed on GnRH neurons in the hypothalamus. Receptor activation induces phospholipase C activity, IP3-mediated calcium release, and ERK phosphorylation. This depolarizes GnRH neurons and triggers action potential firing, driving pulsatile GnRH release into the hypophyseal portal circulation.

Differential Potency vs. KP-10

While kisspeptin-10 (the short 10-residue form) is commonly used in research, KP-54 has a longer plasma half-life (~28 minutes vs. ~3 minutes for KP-10) and greater potency in stimulating LH release, particularly in infertility clinical settings. The longer half-life of KP-54 allows sustained stimulation of GnRH neurons for clinical trigger use.


Research Summary

IVF Trigger

Phase 2

Phase 2 trials evaluated KP-54 as an oocyte maturation trigger in IVF cycles, replacing hCG. A single IV dose of KP-54 induced endogenous LH surges comparable to standard hCG triggers. This approach may reduce ovarian hyperstimulation syndrome (OHSS) risk by producing a self-limiting LH surge rather than sustained hCG exposure.

Hypothalamic Amenorrhea

Phase 2

Women with hypothalamic amenorrhea show significantly reduced kisspeptin signaling. Pulsatile KP-54 infusion restores GnRH pulsatility and LH secretion in these patients. A Phase 2 trial showed subcutaneous KP-54 administration twice daily restored menstrual cycles in women with functional hypothalamic amenorrhea.


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Research Protocols

GoalDoseFrequencyRoute
IVF oocyte trigger9.6 nmol/kg IVSingle bolusIntravenous
Hypothalamic amenorrhea1-5 nmol/kg SCTwice daily x 8 weeksSubcutaneous

Clinical trial use. Not commercially available as standalone product.


Interactions

Downstream
GnRH analogs
Kisspeptin acts upstream of GnRH; effects blocked by GnRH antagonists
Antagonism
RFRP inhibits GnRH neurons where kisspeptin activates them
Feedback
Estradiol regulates kisspeptin neuron activity (both positive and negative feedback)

Safety Profile

KP-54 is well tolerated in clinical trials. Adverse effects include flushing, mild headache, and transient hypotension from vasodilation. No significant ovarian hyperstimulation syndrome (OHSS) in IVF trigger studies. Self-limiting LH surge profile improves the OHSS safety profile versus hCG. No arrhythmias or hepatotoxicity reported.


References

  • [1]Jayasena CN et al. (2014). Subcutaneous injection of kisspeptin-54 accurately induces oocyte maturation and luteinization in women undergoing in vitro fertilization. Journal of Clinical Endocrinology and Metabolism, 99(12), E2406-E2415.
  • [2]Jayasena CN et al. (2014). Twice-weekly kisspeptin-54 administration induces menstrual cycles in hypothalamic amenorrhoea. Clinical Endocrinology, 81(5), 694-701.
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Data Sources & External References
Source: peer-reviewed literature  ·  Domain: ascendpeptide.org
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