📚 Wiki Hormonal & Reproductive Neuropeptide VF

Neuropeptide VF

● Preclinical
Neuropeptide VF (NPVF)
Also known as: RFRP-1 Precursor Peptide, RF-Amide Peptide, GnIH-Related
Page last reviewed

Quick Summary

Neuropeptide VF (NPVF) is a member of the RF-amide neuropeptide family, characterized by a C-terminal Arg-Phe-amide sequence. NPVF and related RFRP peptides are processed from the NPVF gene precursor and serve as mammalian homologs of gonadotropin-inhibitory hormone (GnIH).

Neuropeptide / RF-Amide Preclinical
Neuropeptide VF (NPVF) is a member of the RF-amide neuropeptide family, characterized by a C-terminal Arg-Phe-amide sequence. NPVF and related RFRP peptides are processed from the NPVF gene precursor and serve as mammalian homologs of gonadotropin-inhibitory hormone (GnIH). They are expressed in the hypothalamus and regulate reproduction, stress responses, food intake, and pain signaling through NPFFR1 and NPFFR2 receptors.
Storage Stability
Lyophilized
1–2 years (-20°C)
Reconstituted
~30 days (2–8°C)
Room temp
Avoid

Mechanism of Action

NPFF Receptor Signaling

NPVF-derived peptides activate NPFFR1 and NPFFR2, Gi-coupled GPCRs that inhibit adenylate cyclase and reduce cAMP. In the hypothalamus, RFRP/NPVF signaling inhibits GnRH neurons, reducing LH and FSH secretion. This GnRH inhibitory action is analogous to the avian GnIH system and provides an inhibitory counterbalance to kisspeptin activation of GnRH.

Opioid System Modulation

NPVF and related RF-amide peptides modulate opioid signaling by interacting with opioid receptors and by regulating opioid release. NPFF receptors mediate anti-opioid effects that oppose morphine analgesia and contribute to opioid tolerance. This anti-opioid activity is distinct from the reproductive actions and is mediated through different receptor distributions.


Research Summary

Reproductive Axis Inhibition

Preclinical

RFRP-3 and NPVF suppress the HPG axis by inhibiting GnRH neuron firing and reducing LH pulse frequency in rodents and non-human primates. RFRP/NPVF expression varies with photoperiod, energy status, and stress, suggesting it integrates environmental signals to regulate fertility. NPFFR1 antagonists restore LH pulsatility in hypogonadal rodent models.

Stress and Food Intake

Preclinical

NPVF peptides modulate food intake and energy balance through hypothalamic actions. Central RFRP-3 administration increases food intake in rodents under certain metabolic states, and NPVF expression is regulated by metabolic hormones including leptin/" class="wiki-internal-link">leptin and ghrelin/" class="wiki-internal-link">ghrelin. Stress-induced NPVF expression may link chronic stress to reproductive suppression.


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Research Protocols

GoalDoseFrequencyRoute
GnRH inhibition10-100 nmol ICVSingleIntracerebroventricular
Opioid modulation1-10 nmol ICVSingleIntracerebroventricular

Preclinical research only. No approved therapeutic use for NPVF peptides.


Interactions

Antagonism
Kisspeptin
RFRP/NPVF inhibits while kisspeptin activates GnRH neurons
Anti-opioid
Opioids
NPFF receptors mediate opioid tolerance and oppose morphine analgesia

Safety Profile

NPVF peptides are endogenous hypothalamic neuropeptides. Exogenous administration studies are preclinical. Anti-opioid properties of NPFF receptors are of interest for reducing opioid tolerance. No human clinical data available for NPVF-derived peptides.


References

  • [1]Ubuka T et al. (2012). Identification of mammalian gonadotropin-inhibitory hormone homologs and its impact on the reproductive axis. Frontiers in Endocrinology, 3, 25.
  • [2]Pinilla L et al. (2012). Kisspeptins and reproduction: physiological roles and regulatory mechanisms. Physiological Reviews, 92(3), 1235-1316.
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Verified Scientific Data Last audited:
Data Sources & External References
Source: peer-reviewed literature  ·  Domain: ascendpeptide.org

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