📚 Wiki Antimicrobial & Immune Dermcidin

Dermcidin

● Preclinical
Dermcidin-1L
Also known as: DCD, Sweat AMP, AIDD
Page last reviewed

Quick Summary

Dermcidin is a constitutively expressed antimicrobial peptide produced by eccrine sweat glands of human skin. It is secreted as a precursor and processed in sweat to yield active peptides that protect the skin surface from bacterial colonization.

Antimicrobial Peptide Preclinical
Dermcidin is a constitutively expressed antimicrobial peptide produced by eccrine sweat glands of human skin. It is secreted as a precursor and processed in sweat to yield active peptides that protect the skin surface from bacterial colonization. Dermcidin is unusual among AMPs in being anionic and active at physiological salt concentrations.
Storage Stability
Lyophilized
1–2 years (-20°C)
Reconstituted
~30 days (2–8°C)
Room temp
Avoid

Mechanism of Action

Zinc-Dependent Channel Formation

Dermcidin forms zinc-ion-dependent oligomeric channels in bacterial membranes. Crystal structures reveal a hexameric barrel architecture that inserts into membranes and dissipates the electrochemical gradient. Zinc binding stabilizes the oligomeric state and is required for full antibacterial activity.

Anionic AMP Activity

Unlike most cationic AMPs, dermcidin carries a net negative charge. Its activity at physiological salt concentrations (which inhibit most cationic AMPs) is facilitated by zinc coordination. This allows dermcidin to function effectively in sweat, which contains inhibitory concentrations of salt for many conventional AMPs.


Research Summary

Skin Defense Function

Preclinical

Dermcidin protects skin from S. aureus, E. coli, and Candida albicans under conditions mimicking sweat. Individuals with atopic dermatitis show reduced dermcidin expression, correlating with increased susceptibility to skin infections. This supports its role as a key component of innate cutaneous immunity.

Structural Biology

Preclinical

X-ray crystallography has resolved the hexameric channel structure of dermcidin at 2.5 angstrom resolution. The structure reveals how zinc bridges peptide subunits to form a stable transmembrane pore. This structural insight is being used to design more potent synthetic analogs.


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Research Protocols

GoalDoseFrequencyRoute
Antibacterial activity in sweat1-10 mcg/mLSingleSweat-like buffer
Zinc-dependent channel studies5-20 uMSingleMembrane bilayer

Endogenous peptide; no supplementation protocols established.


Interactions

Required
Zinc ions
Zinc is essential for oligomeric channel assembly and full activity
Synergy
Beta-defensins
Complementary activity in cutaneous innate defense

Safety Profile

Dermcidin is an endogenous human peptide constitutively expressed in sweat glands, suggesting an inherently favorable safety profile at physiological concentrations. It is selective for bacteria over human cells due to its zinc-dependent mechanism. No adverse effects from endogenous production reported.


References

  • [1]Schittek B et al. (2001). Dermcidin: a novel human antibiotic peptide secreted by sweat glands. Nature Immunology, 2(12), 1133-1137.
  • [2]Song C et al. (2013). Crystal structure and functional mechanism of a human antimicrobial membrane channel. PNAS, 110(12), 4586-4591.
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Verified Scientific Data Last audited:
Data Sources & External References
Source: peer-reviewed literature  ·  Domain: ascendpeptide.org

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