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Avexitide

◉ Phase 3
Avexitide
Also known as: Exendin 9-39, GLP-1 Receptor Antagonist, PC-DAC:Exendin-9
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Quick Summary

Avexitide (exendin 9-39) is a competitive antagonist of the GLP-1 receptor derived from a truncated form of the Gila monster peptide exendin-4. It blocks the action of endogenous GLP-1, preventing the exaggerated insulin secretion and hypoglycemia seen in post-gastric bypass hypoglycemia (PGBH) and congenital hyperinsulinism.

Metabolic Peptide / GLP-1 Antagonist Phase 3
Avexitide (exendin 9-39) is a competitive antagonist of the GLP-1 receptor derived from a truncated form of the Gila monster peptide exendin-4. It blocks the action of endogenous GLP-1, preventing the exaggerated insulin secretion and hypoglycemia seen in post-gastric bypass hypoglycemia (PGBH) and congenital hyperinsulinism. The FDA granted it Breakthrough Therapy Designation for PGBH.
Storage Stability
Lyophilized
6–12 months (2–8°C)
Reconstituted
~30 days (2–8°C)
Room temp
Avoid

Mechanism of Action

GLP-1 Receptor Blockade

Avexitide (exendin 9-39) competitively blocks the GLP-1 receptor without activating it. By preventing endogenous GLP-1 from binding, it reduces glucose-dependent insulin secretion and the amplification of insulin release seen postprandially after Roux-en-Y gastric bypass surgery. This directly addresses the hyperinsulinism underlying post-bypass hypoglycemia.

Glucagon Restoration

GLP-1 receptor activation normally suppresses glucagon secretion. Avexitide blockade restores postprandial glucagon release, which counteracts hypoglycemia by stimulating hepatic glucose production. The restoration of the glucagon counterregulatory response is a key component of its therapeutic effect.


Research Summary

Post-Gastric Bypass Hypoglycemia

Phase 3

Phase 2 trials demonstrated that continuous subcutaneous infusion of avexitide significantly reduced hypoglycemic episodes in PGBH patients following gastric bypass surgery. Glucose nadir values improved and time spent in hypoglycemia decreased by approximately 70% versus placebo. Phase 3 trials are ongoing.

Congenital Hyperinsulinism

Phase 2

Avexitide has shown efficacy in diazoxide-unresponsive congenital hyperinsulinism, reducing hypoglycemic episodes in pediatric patients. It represents a non-surgical alternative for patients who would otherwise require near-total pancreatectomy.


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Research Protocols

GoalDoseFrequencyRoute
PGBH treatment30-60 mg/dayContinuous SC infusionSubcutaneous pump
Acute testing0.1-1 pmol/kg/minContinuousIV infusion

FDA Breakthrough Therapy Designation. Not yet approved. Investigational use only.


Interactions

Antagonism
GLP-1 agonists (semaglutide, liraglutide)
Directly opposes GLP-1 agonist action; not combined therapeutically
Additive
Diazoxide
Complementary mechanisms for reducing insulin hypersecretion

Safety Profile

Avexitide is generally well tolerated. Common adverse effects include injection site reactions and mild gastrointestinal symptoms. By blocking GLP-1 receptor signaling, it may increase appetite and food intake as a side effect. Long-term cardiovascular effects of sustained GLP-1 receptor blockade require monitoring given the known cardioprotective role of GLP-1. No serious adverse events identified in Phase 2 trials.


References

  • [1]Craig CM et al. (2017). Efficacy and pharmacokinetics of subcutaneous exendin (9-39) in patients with post-bariatric hypoglycaemia. Diabetes, Obesity and Metabolism, 20(4), 923-930.
  • [2]Salehi M et al. (2012). Hypoglycemia after gastric bypass surgery: current concepts and controversies. Journal of Clinical Endocrinology and Metabolism, 99(7), 2236-2245.
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Verified Scientific Data Last audited:
Data Sources & External References
Source: peer-reviewed literature  ·  Domain: ascendpeptide.org

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