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Secretoneurin

● Preclinical
Secretoneurin
Also known as: SN, Secretogranin II Fragment, CGRP-Like Neuropeptide
Page last reviewed

Quick Summary

Secretoneurin is a 33-amino acid neuropeptide derived from secretogranin II (chromogranin C), one of the granin family proteins stored in neuroendocrine secretory vesicles. It is generated by proteolytic cleavage of secretogranin II and is expressed throughout the central and peripheral nervous system.

Neuropeptide / Neuroendocrine Preclinical
Secretoneurin is a 33-amino acid neuropeptide derived from secretogranin II (chromogranin C), one of the granin family proteins stored in neuroendocrine secretory vesicles. It is generated by proteolytic cleavage of secretogranin II and is expressed throughout the central and peripheral nervous system. Secretoneurin promotes neurite outgrowth, stimulates dopamine release, modulates pain, and has potent angiogenic properties that are being exploited for therapeutic ischemia treatment.
Storage Stability
Lyophilized
1–2 years (-20°C)
Reconstituted
~30 days (2–8°C)
Room temp
Avoid

Mechanism of Action

Dopamine Release Stimulation

Secretoneurin stimulates dopamine release from striatal neurons and dopaminergic terminals in a calcium-dependent manner. This dopaminergic modulation is independent of conventional neurotransmitter release mechanisms and may contribute to motor function regulation. Secretoneurin levels are reduced in Parkinson disease brains, suggesting a role in dopaminergic circuit maintenance.

Angiogenesis

Secretoneurin is a potent promoter of endothelial cell migration, proliferation, and tube formation. Its angiogenic activity is comparable to VEGF at equimolar concentrations. Secretoneurin activates VEGF receptor signaling and mobilizes endothelial progenitor cells from bone marrow. This activity is being exploited in preclinical models of peripheral arterial disease and myocardial ischemia.


Research Summary

Therapeutic Angiogenesis

Preclinical

Intramuscular secretoneurin gene transfer in rodent and porcine models of critical limb ischemia significantly improves limb perfusion, collateral vessel formation, and muscle function. The angiogenic response is comparable to VEGF gene therapy without the edema and vascular leakage side effects associated with VEGF overexpression.

Cardiac Protection

Preclinical

Secretoneurin administration in myocardial infarction models reduces infarct size and improves cardiac function through angiogenic and anti-apoptotic mechanisms. Plasma secretoneurin levels are elevated following acute MI, suggesting a compensatory angiogenic response.


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Research Protocols

GoalDoseFrequencyRoute
Angiogenesis induction1-10 nMSingle / repeatedCell culture / IM injection
Dopamine release0.1-1 nMSingleStriatal superfusion

Preclinical only. No approved human use. Angiogenic gene therapy approach in early clinical development.


Interactions

Synergy
VEGF
Complementary angiogenic mechanisms; combined approaches may be additive
Co-Released
Both granin family members co-secreted from neuroendocrine vesicles

Safety Profile

Secretoneurin is an endogenous neuropeptide with physiological roles in neurotransmitter regulation and tissue homeostasis. Preclinical angiogenic therapy studies show efficacy without significant toxicity. The lack of vascular leakage side effects compared to VEGF is a potential advantage. Human safety data absent.


References

  • [1]Kirchmair R et al. (2004). Secretoneurin, an angiogenic neuropeptide, induces angiogenesis and arteriogenesis. Arteriosclerosis, Thrombosis, and Vascular Biology, 24(10), 1892-1898.
  • [2]Fischer-Colbrie R et al. (1995). GABA, enkephalin/" class="wiki-internal-link">enkephalin and VIP in adrenal chromaffin cells: gene expression, storage and secretion. Journal of the Autonomic Nervous System, 54(1), 61-66.
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Verified Scientific Data Last audited:
Data Sources & External References
Source: peer-reviewed literature  ·  Domain: ascendpeptide.org

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